J Blenis Harvard Medical School

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Dr. John Blenis, Principal Investigator — Blenis Lab

http://www.blenislab.org/john-blenis

Dr. Blenis earned his B. A. from University of California, Berkeley in 1977 and his Ph.D. from Michigan State University in 1983. His postdoctoral research in the Department of …

John Blenis - Professor - Harvard University | LinkedIn

https://www.linkedin.com/in/john-blenis-7a131512

John Blenis Professor at Weill Cornell Medical College Boston, Massachusetts, United States 268 followers 261 connections Join to view profile Harvard University Michigan …

Rapamycin: one drug, many effects - PubMed

https://pubmed.ncbi.nlm.nih.gov/24508508/

Abstract. The mammalian target of rapamycin (mTOR) signaling pathway is a master regulator of cell growth and metabolism. Deregulation of the mTOR pathway has …

John BLENIS | Principal Investigator | Ph.D. | Weill …

https://www.researchgate.net/profile/John-Blenis

John Blenis The alteration of metabolic pathways is a critical strategy for cancer cells to attain the traits necessary for metastasis in disease progression. Here, we find that …

Blenis Lab

http://www.blenislab.org/

The Blenis Laboratory is a part of the Meyer Cancer Center at Weill Cornell Medicine, a newly established cancer center in the heart of NYC. Dr. Blenis heads the Department of …

People | Cell Biology - Harvard University

https://cellbio.hms.harvard.edu/people

Silvia Harper Administrative Coordinator, Dr. Huang 617-432-3132 Email Wade Harper, Ph.D. Bert and Natalie Vallee Professor of Molecular Pathology (HMS) Professor of Cell …

Rapamycin-FKBP specifically blocks growth-dependent …

https://pubmed.ncbi.nlm.nih.gov/1377606/

Abstract. The macrolide rapamycin blocks cell cycle progression in yeast and various animal cells by an unknown mechanism. We demonstrate that rapamycin …

Evidence for two catalytically active kinase domains in …

https://pubmed.ncbi.nlm.nih.gov/8622665/

Abstract Mitogen-activated protein kinase and one of its targets, pp90rsk (ribosomal S6 kinase [RSK]), represent two serine/threonine kinases in the Ras-activated signalling …

The Ras-ERK and PI3K-mTOR pathways: cross-talk and …

https://pubmed.ncbi.nlm.nih.gov/21531565/

The Ras-extracellular signal-regulated kinase (Ras-ERK) and phosphatidylinositol 3-kinase-mammalian target of rapamycin (PI3K-mTOR) signaling pathways are the chief …

Phosphorylation of c-Fos at the C-terminus enhances its …

https://pubmed.ncbi.nlm.nih.gov/8622865/

1 Department of Cell Biology, Harvard Medical School, Boston, Massachusetts 02115, USA. PMID: 8622865 Abstract c-Fos is phosphorylated by MAP …

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